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Clinical Psychopharmacology and Neuroscience  Vol.21 No.1.jpg
SCOPUS 학술저널

The Clinical Observation of Inflammation Theory for Depression: The Initiative of the Formosa Long COVID Multicenter Study (FOCuS)

There is growing evidence that the coronavirus disease 2019 (COVID-19) caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is associated with increased risks of psychiatric sequelae. Depression, anxiety, cognitive impairments, sleep disturbance, and fatigue during and after the acute phase of COVID-19 are prevalent, long-lasting, and exerting negative consequences on well-being and imposing a huge burden on healthcare systems and society. This current review presented timely updates of clinical research findings, particularly focusing on the pathogenetic mechanisms underlying the neuropsychiatric sequelae, and identified potential key targets for developing effective treatment strategies for long COVID. In addition, we introduced the Formosa Long COVID Multicenter Study (FOCuS), which aims to apply the inflammation theory to the pathogenesis and the psychosocial and nutrition treatments of post-COVID depression and anxiety.

INTRODUCTION

CLINICAL MANIFESTATIONS OF LONG COVID PSYCHIATRIC SEQUELAE

DEPRESSION AS A CENTRAL SYMPTOM OF LONG COVID

COULD THE INFLAMMATION THEORY OF DEPRESSION APPLY TO LONG COVID SEQUELAE?

ADDRESSING INFLAMMATION AS A TREATMENT TARGET FOR POST-COVID DEPRESSION

POST-COVID CLINICAL OBSERVATION TO TEST THE INFLAMMATION THEORY OF DEPRESSION: THE FOCUS

CONCLUSION

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